Subject areas:
Research
My research group aims to understand the key mechanisms that dictate the M. tuberculosis-human relationship with the goal of breaking these ties, targeting either host or pathogen factors to eliminate M. tuberculosis infection and, with that, to contribute to solving a steadily increasing threat in global health. In particular, while current vaccine strategies aimed at promoting specific immune responses (e.g. T-cells and cytokines) to various M. tuberculosis antigens have failed, we believe that promoting innate responses of host cell macrophages, the cell type where M. tuberculosis grows, represents an unconventional strategy to block bacterial infection.